If the Urea cycle turns more to process Ammonia, that might make sense! I was definitely having mild headaches during the period which I thought were mainly caused by the elevated Lactate.
My diet was under-caloric the day before and low carb (18g) according to Cronometer. I took .2g of Tryptophan the night before to test the Kynurenic pathway and get a read on B6 status (alongside a plasma test). Plasma B6 was low quartile 5.5ng/mL (2.1-21.7 ng/mL range).
Both XA and KA were reasonably high (barely within normal, but I had only taken .2g instead of the 2g test). So, I am reasonable positive I had lowered B6 function. A possible confounder in the "quantification" of EBV impact given it likely limited pathways the hypothesized EBV modification puts pressure on. (I also have HSV-1, but believe that it's far more limited in activity than EBV)
Hippurate is the only blip, and it could easily be confounded by SIBO, which I definitely had at the time (and, may still have now...though I've gone thru two rounds of anti-microbials since).
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All aside, I'm beginning to think that OAT is a very poor way to quantify one's current HHV state.
It would be nice to have a way to evaluate Ammonia and Lactate levels. Lactate in the blood is possible, but pricey. And, the Ammonia tools available seem to be more calibrated to monitoring fish tanks...probably too inaccurate.
