Overview of the Protocol

This disease model this protocol is based on describes the downstream effects of various blind spots in our immune system which further explain why some health problems become chronic and/or develop into progressive diseases.

This understanding reveals how some harmful microorganisms can hide within protective shields called "biofilms" throughout our bodies—in places like our nose, mouth, lungs, digestive tract, reproductive organs, and even our bloodstream. (Other microorganisms may also hide in parasitic "cysts" and fibrin "nests".)

These biofilms begin forming during childhood, typically growing slowly over many years. Collected data suggests this gradual buildup might be connected to what we consider as natural aging processes. However, when the immune system faces challenges from infections, medical procedures, medications, injuries, poor nutrition, or ongoing stress, these biofilms can grow much more rapidly.

In a healthy body, the immune system keeps biofilms in check by detecting and attacking pathogenic microorganisms when they venture outside these protected areas. A sufficiency of beneficial "probiotic" species can also degrade biofilms and attack pathogens.

When this defense system becomes compromised, the biofilms can increase their surface area and some of these hidden microorganisms then produce an increasing amount of harmful substances, particularly a toxin called acetaldehyde—the same compound produced when your body processes alcohol.

This toxin disrupts cellular energy production and damages both your immune system and the lining of your digestive tract. A damaged gut lining allows more harmful microorganisms to cross this critical barrier, triggering an immune response aimed at eliminating these invaders. The resulting long-term inflammation and hypoxia can transform temporary mineral shortages into serious, system-wide deficiencies that are difficult to detect and correct.

One of the challenges in addressing this condition is its cyclic nature—symptoms often improve and worsen in waves. This pattern, combined with inhibited nutrient absorption in the small intestine, makes it extremely difficult to correct mineral deficiencies through diet or oral supplements alone. Research shows that homeostasis for up to eleven metals may be affected at various times, depending on the state of inflammation.

During chronic inflammation, accurately measuring systemic mineral levels becomes very difficult, because inflammatory signalling changes how minerals move across cell membranes throughout different tissues in the body. Some tissues will store higher amounts of minerals, eg. brain, liver and kidneys, while other tissues will be deficient. Measuring mineral status via serum or red blood cells in this state becomes highly problematic.

Recent studies have uncovered that disruption of key immune regulators, along with ongoing inflammation involving a hormone called hepcidin, disturbs the body's balance of many biologically essential metals. These minerals rely on shared transportation systems in our cells called divalent metal transporter 1 and ferroportin. When the mineral status is disrupted, it creates a cascade of additional problems throughout the body.

We have further observed that other minerals become depleted in this disease model. For example, silicon may be lost when it combines with acetaldehyde produced by microorganisms. Additionally, food intolerances / dietary restrictions and toxins from bacteria or fungi can disrupt zinc and other mineral levels in the body.

When certain minerals become deficient, it affects numerous bodily functions. For instance, shortages of minerals like lithium and rubidium can worsen brain chemistry and kidney function. Impaired kidneys then lead to electrolyte imbalances that disrupt energy production, nerve function, immune activity, and muscle contractions—including those of the heart. This creates a progressive array of cycles which further weaken the immune system and severely disrupts the beneficial microbe communities throughout the body.

The resulting chain reaction affects numerous bodily systems: our immune system becomes dysregulated, brain chemical balances shift, stress hormone levels change, vitamin metabolism alters, cholesterol production fluctuates, and regulation of histamine, hormones, kidney function, and even body temperature becomes compromised.

Early testing of this experimental treatment protocol has shown encouraging results. Some individuals have achieved long-term remission, while others are showing significant improvement on their path to recovery. Formal clinical trials are now being organised to study these effects more systematically.

This pattern of metabolic disruption appears in many infections and chronic health conditions. Associated problems include dysregulated innate immune response, cellular energy production issues, low oxygen levels within cells, nervous system dysfunction, histamine regulation problems, and challenges in producing collagen.

As research continues, we are finding that patterns of deficiencies can help predict various disease features and their severity. This growing understanding is continuously improving treatment approaches, offering new hope for those suffering from these challenging conditions.

The intervention protocol continues to evolve as we uncover more about how these complex systems interact and how homeostasis can be restored more efficiently.